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In this study, VDR is essential for the morphogenetic processes that drive hair cycle progression and the cell division within the hair follicle. We identify Vdr as an important regulator of catagen by showing that the phenotype associated with Vdr cKO is the result of diminished apoptosis of epithelial strands of epithelial cells that have been killed in the HF during the catagen phase. The delay in the entry of the HF to anagen results in alopecia, and the HF turns into a dermal cyst composed of epidermal cells.

To investigate the role of VDR in the regulation of apoptosis in catagen, we carried out a comprehensive screen for putative VDR targets using functional annotation enrichment analysis. In comparison to control CO fibroblasts, there were 268 genes that are regulated by the unliganded VDR www.dcdataroom.com/what-is-data-room-due-diligence/ and downregulated by activation dependent on 1,25D. The majority of these genes are also upregulated by fibroblasts from WT.

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